Collagen Vascular Disease Sebagai Faktor Risiko Terjadinya Mata Kering

Main Author: Eka Purwanti, Mutya
Format: Article info application/pdf Journal
Bahasa: eng
Terbitan: Rifa' Institute , 2022
Subjects:
Online Access: https://fusion.rifainstitute.com/index.php/fusion/article/view/134
https://fusion.rifainstitute.com/index.php/fusion/article/view/134/133
ctrlnum article-134
fullrecord <?xml version="1.0"?> <dc schemaLocation="http://www.openarchives.org/OAI/2.0/oai_dc/ http://www.openarchives.org/OAI/2.0/oai_dc.xsd"><title lang="en-US">Collagen Vascular Disease Sebagai Faktor Risiko Terjadinya Mata Kering</title><creator>Eka Purwanti, Mutya</creator><subject lang="en-US">collagen vascular disease</subject><subject lang="en-US">dry eyes</subject><subject lang="en-US">autoimmune</subject><description lang="en-US">Dry eye is one of the eye diseases that is the most common reason a person visits an ophthalmologist. Globally, the prevalence of dry eye with or without symptoms ranges from 5-50%. Dry eye is more common in women than men and increases with age. Dry eye is a disease that has many similarities to autoimmune diseases. One of the autoimmune diseases that can be a high risk factor for dry eyes is collagen vascular disease. The mechanism of dry eye in patients with collagen vascular disease is thought to occur due to the role of muscarinic acetylcholine receptor type 3 autoantigens and B cells as APCs that produce autoantibodies. This autoimmune process causes a loss of homeostasis due to the presence of inflammatory mediators that damage the lacrimal functional unit. Dry eyes that occur due to collagen vascular disease are characterized by symptoms of irritation and discomfort in the eyes. Currently, knowledge about the pathogenesis and treatment is still growing.</description><publisher lang="en-US">Rifa' Institute</publisher><date>2022-01-20</date><type>Journal:Article</type><type>Other:info:eu-repo/semantics/publishedVersion</type><type>Journal:Article</type><type>File:application/pdf</type><identifier>https://fusion.rifainstitute.com/index.php/fusion/article/view/134</identifier><identifier>10.54543/fusion.v2i01.134</identifier><source lang="en-US">Jurnal Syntax Fusion; Vol 2 No 01 (2022): Jurnal Syntax Fusion: Jurnal Nasional Indonesia; 87-93</source><source lang="id-ID">Jurnal Syntax Fusion; Vol 2 No 01 (2022): Jurnal Syntax Fusion: Jurnal Nasional Indonesia; 87-93</source><source>2775-6440</source><source>2808-7208</source><source>10.54543/fusion.v2i01</source><language>eng</language><relation>https://fusion.rifainstitute.com/index.php/fusion/article/view/134/133</relation><rights lang="en-US">Copyright (c) 2022 Jurnal Syntax Fusion</rights><recordID>article-134</recordID></dc>
language eng
format Journal:Article
Journal
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File:application/pdf
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Journal:Journal
author Eka Purwanti, Mutya
title Collagen Vascular Disease Sebagai Faktor Risiko Terjadinya Mata Kering
publisher Rifa' Institute
publishDate 2022
topic collagen vascular disease
dry eyes
autoimmune
url https://fusion.rifainstitute.com/index.php/fusion/article/view/134
https://fusion.rifainstitute.com/index.php/fusion/article/view/134/133
contents Dry eye is one of the eye diseases that is the most common reason a person visits an ophthalmologist. Globally, the prevalence of dry eye with or without symptoms ranges from 5-50%. Dry eye is more common in women than men and increases with age. Dry eye is a disease that has many similarities to autoimmune diseases. One of the autoimmune diseases that can be a high risk factor for dry eyes is collagen vascular disease. The mechanism of dry eye in patients with collagen vascular disease is thought to occur due to the role of muscarinic acetylcholine receptor type 3 autoantigens and B cells as APCs that produce autoantibodies. This autoimmune process causes a loss of homeostasis due to the presence of inflammatory mediators that damage the lacrimal functional unit. Dry eyes that occur due to collagen vascular disease are characterized by symptoms of irritation and discomfort in the eyes. Currently, knowledge about the pathogenesis and treatment is still growing.
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